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Rubin's Pathology: Clinicopathologic Foundations Of Medicine 7th Edition By David S. Strayer; Emanuel Rubin Md| All Chapters 1-34 Covered| Verified| Rated A+| Latest Edition| Test Bank€17,81
Rubin's Pathology: Clinicopathologic Foundations Of Medicine 7th Edition By David S. Strayer; Emanuel Rubin Md| All Chapters 1-34 Covered| Verified| Rated A+| Latest Edition| Test Bank
Rubin's Pathology: Clinicopathologic Foundations Of Medicine 7th Edition By David S. Strayer; Emanuel Rubin Md| All Chapters 1-34 Covered| Verified| Rated A+| Latest Edition| Test Bank
Table Of Contents
SECTION I: MECHANISMS OF DISEASE
Chapter 1: Cell Adaptation, Injury And Death
Chapte...
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Voorbeeld van de inhoud
Rubin's Pathology: Clinicopathologic Foundations Of
Medicine 7th Edition By David S. Strayer; Emanuel Rubin
Md| All Chapters 1-34 Covered| Original| Verified|
Rated A+| Latest Edition| Test Bank
, Table Of Contents
SECTION I: MECHANISMS OF DISEASE
Chapter 1: Cell Adaptation, Injury And Death
Chapter 2: Inflammation
Chapter 3: Repair, Regeneration And Fibrosis
Chapter 4: Immunopathology
Chapter 5: Neoplasia
Chapter 6: Developmental And Genetic Diseases
Chapter 7: Hemodynamic Disorders
Chapter 8: Environmental And Nutritional Pathology
Chapter 9: Infectious And Parasitic Diseases
SECTION II: PATHOGENESIS OF SYSTEMIC CONDITIONS
Chapter 10: Aging
Chapter 11: Systemic Autoimmune Diseases
Chapter 12: Sepsis
Chapter 13: Obesity And Diabetes Mellitus
Chapter 14: The Pathology Of Pregnancy
Chapter 15: The Amyloidoses
SECTION III: DISEASES OF INDIVIDUAL ORGAN SYSTEMS
Chapter 16: Blood Vessels
Chapter 17: The Heart
Chapter 18: The Respiratory System
Chapter 19: The Gastrointestinal Tract
Chapter 20: The Liver And Biliary System
Chapter 21: The Pancreas
Chapter 22: The Kidney
Chapter 23: The Lower Urinary Tract And Male Reproductive System
,Chapter 24: The Female Reproductive System And Peritoneum
Chapter 25: The Breast
Chapter 26: Hematopathology
Chapter 27: The Endocrine System
Chapter 28: The Skin
Chapter 29: The Head And Neck
Chapter 30: Bones, Joints And Soft Tissue
Chapter 31: Skeletal Muscle And Peripheral Nervous System
Chapter 32: The Central Nervous System
Chapter 33: The Eye
Chapter 34: Forensic Pathology
,SECTION I: MECHANISMS OF DISEASE
Rubin's Pathology: Clinicopathologic Foundations Of Medicine 7th Edition
Chapter 1: Cell Adaptation, Injury And Death
Multiple Choice
1. Ischemia And Other Toxic Injuries Increase The Accumulation Of
Intracellular Calcium As A Result Of:
A) Release Of Stored Calcium From The Mitochondria.
B) Improved Intracellular Volume Regulation.
C) Decreased Influx Across The Cell Membrane.
D) Attraction Of Calcium To Fatty Infiltrates.
Answer: A)
Feedback:
Ischemia And Toxic Injuries Lead To Impaired Cell Function, Causing The
Release Of Calcium Stored In The Mitochondria And Other Organelles. This
Results In An Increase In Intracellular Calcium Levels, Which Can Activate
Enzymes That Damage Cell Structures, Contributing To Cell Injury.
2. The Patient Is Found To Have Liver Disease, Resulting In The Removal Of
A Lobe Of His Liver. Adaptation To The Reduced Size Of The Liver Leads To
_ Of The Remaining Liver Cells.
A) Metaplasia
B) Organ Atrophy
C) Compensatory Hyperplasia
,D) Physiologic Hypertrophy
Answer: C)
Feedback:
Compensatory Hyperplasia Refers To The Process Where The Remaining
Cells Increase In Size Or Number To Compensate For The Lost Tissue. This
Is A Normal Response In Cases Where An Organ Is Partially Removed Or
Damaged.
3. A Person Eating Peanuts Starts Choking And Collapses. His Airway
Obstruction Is Partially Cleared, But He Remains Hypoxic Until He Reaches
The Hospital. The Prolonged Cell Hypoxia Caused A Cerebral Infarction And
Resulting _ In The Brain.
A) Caspase Activation
B) Coagulation Necrosis
C) Rapid Phagocytosis
D) Protein P53 Deficiency
Answer: B)
Feedback:
Coagulation Necrosis Is A Form Of Cell Death That Typically Results From
Ischemic Injuries, Such As Those Seen In Cerebral Infarctions. This Type Of
Necrosis Is Characterized By The Preservation Of The Tissue Structure,
Even Though The Cells Within The Tissue Die.
4. Bacteria And Viruses Cause Cell Damage By _, Which Is Unique From The
Intracellular Damage Caused By Other Injurious Agents.
A) Disrupting The Sodium/Potassium ATPase Pump
,B) Interrupting Oxidative Metabolism Processes
C) Replicating And Producing Continued Injury
D) Decreasing Protein Synthesis And Function
Answer: C)
Feedback:
Unlike Other Injurious Agents, Bacteria And Viruses Damage Cells By
Replicating Within Them, Leading To Ongoing Harm As The Pathogen
Reproduces And Spreads. This Continuous Damage Is A Hallmark Of
Infection-Related Cell Injury.
5. The Patient Has A Prolonged Interruption In Arterial Blood Flow To His
Left Kidney, Causing Hypoxic Cell Injury And The Release Of Free Radicals.
Free Radicals Damage Cells By:
A) Destroying Phospholipids In The Cell Membrane.
B) Altering The Immune Response Of The Cell.
C) Disrupting Calcium Storage In The Cell.
D) Inactivation Of Enzymes And Mitochondria.
Answer: A)
Feedback:
Free Radicals Can Damage Cell Membranes By Peroxidizing Phospholipids,
Leading To Increased Membrane Permeability And Loss Of Cellular
Integrity. This Contributes To Cellular Injury, Especially In Ischemic
Conditions.
6. Injured Cells Have Impaired Flow Of Substances Through The Cell
Membrane As A Result Of:
, A) Increased Fat Load.
B) Altered Permeability.
C) Altered Glucose Utilization.
D) Increased Surface Receptors.
Answer: B)
Feedback:
Cellular Injury Often Results In Changes To The Cell Membrane's
Permeability, Impairing The Flow Of Essential Substances Into And Out Of
The Cell. This Can Lead To Swelling, Ion Imbalances, And Further Damage.
7. Reversible Adaptive Intracellular Responses Are Initiated By:
A) Stimulus Overload.
B) Genetic Mutations.
C) Chemical Messengers.
D) Mitochondrial Dna.
Answer: C)
Feedback:
Reversible Adaptive Responses In Cells, Such As Hypertrophy Or
Hyperplasia, Are Often Initiated By Chemical Signals (E.G., Hormones,
Growth Factors) That Help The Cell Adjust To Stress Or Injury Without
Permanent Damage.
8. Injured Cells Become Very Swollen As A Result Of:
A) Increased Cell Protein Synthesis.
B) Altered Cell Volume Regulation.
C) Passive Entry Of Potassium Into The Cell.
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