Mb ascp f Study guides, Class notes & Summaries
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MB (ASCP) Exam Questions and Answer
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MB (ASCP) Exam Questions and Answers 
Predominant form of DNA Right-handed B 
DNA and histone interaction is due to what type of bond? Ionic 
What stains to the minor groove of DNA duplex? SYBR Green 
What is the most abundant type of RNA? Ribosomal RNA (rRNA) 
What does RNA act as in a retrovirus? Genome and mRNA 
What increases half-life of mRNA? 5' Methyl Cap 
What kind of DNA is most likely to be transcriptionally active? Euchromatin 
What is the most common type of eukaryotic DNA? ...
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MB ASCP FINAL EXAM 2024 QUESTIONS & 100% CORRECT ANSWERS
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MB ASCP FINAL EXAM 2024 QUESTIONS & 100% CORRECT ANSWERS 
Here You'll Find Questions & Answers Such As: 
What is the function of tRNA? - Answer - Carries aa to ribosome 
Anticodon pairs with codon on mRNA strand 
 
Feedback inhibition - Answer - Product of pathway is noncompetitive inhibitor 
Binds to allosteric site to slow down rxn b/c too much product
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MB ASCP FINAL EXAM QUESTION & ANSWERS 2024
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MB ASCP FINAL EXAM QUESTION & ANSWERS 2024
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MB ASCP FINAL EXAM Questions with Correct Answers Already Passed Graded A+ 2024.pdf
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MB ASCP FINAL EXAM Questions with Correct Answers Already Passed Graded A+
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ASCP MB: Extra-Practice Questions and Answers Scored A
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ASCP MB: Extra-Practice Questions and Answers Scored A The Klenow fragment of E. coli DNA polymerase I is used: to label DNA probes by random primer labeling. 
What is a solution hybridization method for the detection of nucleic acid:protein interactions? gel mobility shift assay 
_____ involves mixing a labeled oligonucleotide with a protein extract to see if a specific interaction occurs, visible with an apparent increase in molecular size on a gel due to the protein:nucleic acid interaction. ...
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MB ASCP Final Exam Practice Questions and Answers Part III (Already Graded A+ - 2023)
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MB ASCP Final Exam Practice Questions and Answers Part III (Already Graded A+ - 2023) Neurofibromatosis 2: tumor suppressor 22q12 missense, frameshift, splice 
Growth regulator mutation. 
66. Neurofibromatosis 1: tumor suppressor 17q11.2 missense, frameshift, splice 
Growth regulator mutation. 
67. Patau: trisomy 13 
68. Phenylketonuria: phenylalanine hydroxylase 
12q24.1 , many mutations 
69. Prader Willi syndrome: chromosome 15 from father 
70. Prothrombin: Chromosome 11, G20210A 
71. Chromoso...
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