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Study Guide: Summary for Acquired Cardiovascular Disease in Cats

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Summary document going through these learning outcomes: Students should be able to: 1. list the common cardiac conditions of the cat 2. discuss the comparative incidence of different feline cardiac diseases 3. comment on prognostic factors in feline myocardial diseases 4. discuss the difficulties of treating asymptomatic hypertrophic cardiomyopathy 5. describe an approach to managing acute congestive heart failure in the cat 6. outline the options for prevention of aortic thromboembolism

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Uploaded on
January 26, 2025
Number of pages
7
Written in
2024/2025
Type
Summary

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Acquired CVS Diseases in Cat 🐈 Restrictive
dilated

L Ventricles are stiff and doesn’t fill
Students should be able to properly + big left atrium
1.​ list the common cardiac conditions of the cat Arrhythmogenic Replacement of the right and regular free
2.​ discuss the comparative incidence of different feline RV wall with fibrous +/ fatty tissue
cardiac diseases →arrhythmias
3.​ comment on prognostic factors in feline myocardial
diseases Unclassified Smth that doesn’t fit
4.​ discuss the difficulties of treating asymptomatic
hypertrophic cardiomyopathy
Echocardiogram Classification
5.​ describe an approach to managing acute congestive
heart failure in the cat
6.​ outline the options for prevention of aortic
thromboembolism

Which heart diseases occur in Cats?
1.​ Cardiomyopathies (most most common)
HCM = walls of LV thicker than they should be
2.​ Congenital diseases
DCM = thin walls that aren’t contracting well
3.​ Arrhythmias
RCM = abnormal fibrous bands crossing the LV
Traditional Cardiomyopathy classification: ARVC = bigger right heart than left heart
UCM = abnormal




Hypertrophic Thick left ventricular walls

Dilated Walls are thinner/normal chambers are

, -​ HCM phenotype = most cats start with increased
LV thickness → as disease progress wall may get
thinner bc death of myocardium → scar tissue →
not contract well = overlap with DCM
-​ Called End-stage HCM
-​ Wall thinning and poor
contraction
-​ White arrows = possible causes
-​ If not these causes = genetic
-​ Mutations
-​ Maine coons/Ragdolls have mutations of
myosin binding protein C

HCM - Hypertrophic Cardiomyopathy
-​ Prevalent in 15% of cats
-​ Not always associated with poor prognosis
-​ Underlying cause
-​ Not from extra “work”
-​ Such as chronic hypertension
-​ True HCM = genetic (mutation)
-​ In cats with HCM
-​ Random orientation of myocytes and myocardial
fibres
-​ From mutation of contractile proteins
-​ VS healthy cats = parallel orientation of fibres
-​ = histopathological abnormalities
-​ Most common cardiomyopathy = HCM
-​ Overlaps with RCM
-​ LV wall thickness + dilation of the left
atrium = RCM phenotype but might look
like HCM
-​ DCM some overlap with ARVC
-​ Unclassified doesn’t fit with any categories
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