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NUR 2063 Essentials of Pathophysiology (NUR2063) / Pathophysiology Final Rasmussen University Winter 2021 CA$22.37   Add to cart
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NUR 2063 Essentials of Pathophysiology (NUR2063) / Pathophysiology Final Rasmussen University Winter 2021

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  • NUR 2063 Essentials Of Pathophysiology (NUR2063)
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  • RASMUSSEN UNIVERSITY

NUR 2063 Essentials of Pathophysiology (NUR2063) / Pathophysiology Final Rasmussen University Winter 2021 S - The Marketplace to Buy and Sell your Study Material Downloaded by: Richarestqueen | Distribution of this document is illegal S - The Marketplace to Buy and Sell your Study Material 5...

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  • May 28, 2022
  • 53
  • 2020/2021
  • Exam (elaborations)
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  • rasmussen college
  • nur2063
  • nur 2063
  • nur 2063 essentials of pathophysiology nur2063
  • nur 2063 essentials of pathophysiology
  • essentials of pathophysiology nur2063
  • essentials of pathophysiology

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  • RASMUSSEN UNIVERSITY
  • NUR 2063 Essentials Of Pathophysiology (NUR2063)
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NUR 2063 Essentials of Pathophysiology
(NUR2063) / Pathophysiology Final
Rasmussen University Winter 2021


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,Pathophysiology Final Rasmussen University Winter 2021 |
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NUR 2063 Essentials of Pathophysiology (NUR2063) /
Pathophysiology Final Rasmussen University Winter 2021


1. •Neither recessive nor dominant—co-dominant.
•Hemoglobin S causes erythrocytes to be abnormally shaped.
•Abnormal erythrocytes carry less oxygen and clog vessels, causing hypoxia and tissue ischemia.


Exercise-induced asthma


Chronic Bronchitis


Sickle Cell Anemia


Occupational asthma



2. blood flow reestablished to quickly


Distributive shock


Anaphylactic shock


Reprofusion injury


spinal cord injury



3. •Life-threatening complication of many conditions
•Results from an inappropriate immune response
•Widespread coagulation followed by massive bleeding because of the depletion of clotting factors


•Complications: shock and multisystem organ failure


Disseminated intravascular coagulation - DIC


Chronic Bronchitis Manifestations and Treatment


Disseminated Intravascular Coagulation DIC


Traumatic Brain Injury Complications



4. thyroid gland - T3, T4, calcitonin
parathyroid gland - parathyroid hormone


during acute respiratory failure



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stress incontinence


secondary brain injury


Glands that regulate calcium



5. altered level of consciousness
BP drops
HR goes up
hypotensive


Glands that regulate calcium


spinal cord injury


shock sighs and symptoms


Reprofusion injury



6. chest pain that occurs while a person is at rest and not exerting himself


does not get better


hemorrhagic stroke


Bone electrolytes


sickle cell crisis


unstable angina



7. Abnormally low white blood cell count


•Normal range = 5,000 to 10,000 cells/mL3 blood


beta cells


Leukopenia


Pneumonia


CT scan



8. damage to the alveoli
air can get in, but not out


emphysema lecture



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Normal CO2 levels


anemia symptoms


Stabilize the patient



9. Manifestations


•May be vague and develop slowly, or may be sudden and severe.
•Symptoms may improve and then suddenly worsen.
•The outward appearance of the head is not an indication of the injury severity.


•Not being able to recall event details
•Indications of a concussion
•Changes in or unequal pupil size
•Seizures
•Asymmetrical facial features


•Fluid draining from the nose, mouth, or ears
•Fracture of skull or face
•Bruising of face
•Swelling at site of injury
•Scalp wound
•Impaired hearing, smell, taste, speech, or vision


•Inability to move one or more limbs
•Irritability (especially in children)
•Personality changes
•Unusual behavior
•Loss of consciousness


•Bradypnea
•Hypotension
•Restlessness
•Lack of coordination
•Lethargy
•Stiff neck
•Vomiting


Acute Respiratory Distress Syndrome (ARDS) Manifestations and Treatment


Acute Bronchitis Manifestations


Tuberculosis Manifestations and Treatment


Traumatic Brain Injury Manifestations




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