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Advanced Patho NURS 5315 UTA Exam 1 Questions and Verified Answers (2024 / 2025) 100% Guarantee Pass
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  • NURS 5315 Advanced Pathophysiology
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  • University Of Texas - Arlington

Advanced Patho NURS 5315 UTA Exam 1 Questions and Verified Answers (2024 / 2025) 100% Guarantee Pass Atrophy: Etiology. Cells decrease in size Patho. Still functional Physiologic: thymus gland in early childhood Pathological: disuse 2. Hypertrophy: E. Increase in cell size P. Increased workloa...

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  • October 25, 2024
  • 15
  • 2024/2025
  • Exam (elaborations)
  • Questions & answers

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  • advanced patho nurs 5315 uta exam 1 questions

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  • University Of Texas - Arlington
  • NURS 5315 Advanced Pathophysiology
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Advanced Patho NURS 5315 UTA Exam 1 Questions & Verified Corre... file:///C:/Users/HP/Desktop/New%20folder%20(4)/Advanced%20P




Advanced Patho NURS 5315 UTA Exam 1

Questions & Verified Correct Answers




1. Atrophy: Etiology. Cells decrease in size Patho.

Still functional

Physiologic: thymus gland in early childhood Pathological:

disuse

2. Hypertrophy: E. Increase in cell size

P. Increased workload Physiologic:

weightlifting

Pathologic: cardiomegaly from HTN

3. Hyperplasia: E. Increase in cell number

P. Increased cellular division

Physiologic: liver regeneration

Pathologic: endometrial- usually r/t hormones
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1 of 15 10/24/2024, 7:35

,Advanced Patho NURS 5315 UTA Exam 1 Questions & Verified Corre... file:///C:/Users/HP/Desktop/New%20folder%20(4)/Advanced%20P




4. Dysplasia: E. Cells change in size, shape, organization

P. AKA atypical hyperplasia, a disorderly proliferation

Physiologic: N/A

Pathologic: squamous dysplasia of cervix from HPV

5. Metaplasia: E. one cell type replaced with another

P. reprogramming of stem cells, reversible Physiologic: N/A

(Not Normal)

Pathologic: stratified squamous cells in bronchial lining r/t cigarette smoke in Lung Cancer,

Esophageal Metaplasia from GERD

6. Hypoxia injury: E. inadequate oxygenation of tissues

P. decrease in mitochondrial function, decreased production of ATP increases anaer- obic

metabolism. eventual cell death.

C.M. hypoxia, cyanosis, cognitive impairment, lethargy

7. Free radical and ROS: E. normal byproduct of ATP production, will overwhelm the

mitochondria- exhaust intracellular antioxidants

P. lipid peroxidation, damage proteins, fragment DNA

C.M. development in Alzheimer's, heart disease, Parkinson's disease, Amyotrophic Lateral

Sclerosis
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