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TEST BANK FORVDAVIS ADVANTAGE FOR PATHOPHYSIOLOGY 2NDEDITION BY CAPRIOTTI
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TEST BANK FORVDAVIS ADVANTAGE FOR PATHOPHYSIOLOGY 2NDEDITION BY CAPRIOTTI
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TEST BANK For Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 2nd Edition by Theresa M Capriotti | Verified Chapter's 1 - 46 | Complete
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COMPLETE TEST BANK FOR DAVIS ADVANTAGE FOR PATHOPHYSIOLOGY: INTRODUCTORY CONCEPTS AND CLINICAL PERSPECTIVES SECOND EDITION BY THERESA CAPRIOTTI DO MSN CRNP RN ||REVISED EDITION
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Test bank for Davis Advantage for Pathophysiology Introductory Concepts and Clinical Perspectives 2nd Edition by Theresa M Capriotti | LATEST UPDATE 2024
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YourVreadingsVinVchapterV9VthisVweekVandVtheVhandoutVdiscussingVtheVriskVcompaniesVfaceVoverVunstructuredVdataVraiseVconcernsVthatVneedVtoVbeVaddressedVwhenVaVcompanyVusesVsocialVm
edia.VWithVyourVindustryVinVmind,VidentifyVandVdiscussVtheseVconcernsVandVaddressVhowV
theyVcanVbeVeffectivelyVhandled
, DAVISVADVANTAGEVFORVPATHOPHYSIOLOGYV2NDVEDITIONVCAPRIOTTIVTESTVBANK
Chapter 1, The Cell in Health and Illnes
V V V V V V V
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TEST BANK FOR DAVIS ADVANTAGE FOR PATHOPHYSIOLOGY 2ND EDITION BY
V V V V V V V V V
CAPRIOTTI V
NURSING TEST BANK V V
MultipleVChoice
IdentifyVtheVchoiceVthatVbestVcompletesVtheVstatementVorVanswersVtheVquestion.
V VV 1.VWhichVstatementVregardingVtheVsodium–potassiumVpumpVisVcorrect?
1. TheVcell’sVplasmaVmembraneVisVmoreVsolubleVtoVsodiumVionsVthanVpotassiumVions.
2. TheVconcentrationVofVsodiumVionsVshouldVbeVhigherVinsideVtheVcellVcompartment.
3. TheVconcentrationVofVpotassiumVionsVshouldVbeVhigherVoutsideVtheVce
llVcompartment.
4. TheVactiveVtransportVinvolvesVpumpingVoutVthreeVsodiumVionsVandVpumpingVinVt
woVpotassiumVions.
V
2.VInVtheVabsenceVofVoxygen,VwhichVcellularVfunctionVcreatesVtheVsameVamountVofVenergyVasVisV
VV
createdVinVtheVpresenceVofVoxygen?
1. DissipationVofVpyruvicVacid
2. InitiationVofVtheVcitricVacidVcycle
3. ActivationVofVacetyl-coenzymeVA
4. CreationVofVacidosisVviaVlacticVacid
3. HowVmanyVadenosineVtriphosphatesV(ATPs)VareVproducedVinVaerobicVenergyVmetabolism?
1.VVV 2
2.VVV 3
3.VVV 3
NURSINGTB.COM
4
4.VVV 5
3
4. WhichVcellVorganellesVdifferVinVtheirVnumberVaccordingVtoVtheVcell’sVenergyVneeds?
1. Ribosomes
2. Mitochondria
3. RibonucleicVacids
4. DeoxyribonucleicVacids
5. WhichVoptionVbestVsupportsVtheVreasonVmoreVenergyVisVproducedVwhenVaVpersonVisVexercising?
1. ExerciseVcausesVanVincreaseVinVtheVsynthesisVofVprotein.
2. ThereVisVanVincreaseVinVtheVproductionVofVpyruvicVacidVinVtheVcells.
3. TheVconversionVofVpyruvicVacidVtoVlacticVacidVisVincreasedVbyVexercise.
4. MuscleVcellsVhaveVmoreVmitochondriaVtoVmeetVenergyVdemands.
6. WhenVdoesVribosomalVproteinVsynthesisVcease?
1. DuringVendoplasmicVreticulumVstress
2. DuringVtheVsynthesisVofVadenosineVtriphosphateV(ATP)
3. DuringVaVsevereVhypoxicVstate
4. DuringVtheVprocessingVofVprohormone
, DAVISVADVANTAGEVFORVPATHOPHYSIOLOGYV2NDVEDITIONVCAPRIOTTIVTESTVBANK
7. WhichVcellularVorganellesVareVresponsibleVforVpropellingVmucusVandVinhaledVdebrisVoutVofVtheVlungs?
1. Cilia
2. Microfilaments
3. SecretoryVvesicles
, DAVISVADVANTAGEVFORVPATHOPHYSIOLOGYV2NDVEDITIONVCAPRIOTTIVTESTVBANK
4. EndoplasmicVreticula
V 8.V WhichVareVtheVkeyVproteinsVinVtheVcontractileVunitsVofVtheVmuscleVcells?
VV
1. ActinVandVmyosin
2. ProhormoneVandVtubulin
3. TubulinVandVactin
4. MyosinVandVprohormone
V 9.V WhichVdeficiencyVcausesVTay–SachsVdisease?
VV
1. Proteasome
2. Peroxisome
3. Macrophage
4. LysosomalVenzymes
V 10.VWhichVisVaVcharacteristicVofVadrenoleukodystrophy?
1. AccumulationVofVganglioside
2. CessationVofVribosomalVproteinVsynthesis
3. AccelerationVofVcellularVproteasomeVactivity
4. AccumulationVofVlong-chainVfattyVacidsVinVtheVnervousVsystem
V 11.VWhichVstatementVregardingVendoplasmicVreticulumV(ER)VstressVisVcorrect?
1. DuringVERVstress,VproteinsVareVrapidlyVdegraded.
2. DuringVERVstress,VlipidsVcannotVtravelVtoVtheirVproperVintracellularVlocations.
3. DuringVERVstress,Vlong-chainVfattyVacidsVaccumulateVinVtheVnervousVsystem.
4. DuringVERVstress,VnondeNgUraR
deSdIsN
ubGsT
taB
nc.eC
sVO
acMcumulateVinVtheVcells.
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12.VAVclientVisVdiagnosedVwithVtypeV1VdiabetesVmellitus.VAtVaVcellularVlevel,VwhichVfunctionVisVli
kelyVtoVbeVinvolved?
1. InabilityVofVribosomesVtoVproduceVaVspecificVtypeVofVprotein
2. IncorrectVprocessingVofVaVproteinVbyVtheVGolgiVapparatus
3. StagnationVofVaVpreviouslyVdynamicVactionVinVmicrotubules
4. ObstructionVofVtheVsmoothVendoplasmicVreticulum
V
13.VAVnewbornVpatientVexhibitsVcharacteristicsVofVsevereVphysicalVdeformities.VWhichVcellularVco
mponentVisVexaminedVtoVdetermineVtheVcauseVandVprobabilityVofVtheVdiseaseVbeingVgeneticallyVtrans
ferred?
1. TransferVRNA
2. RibosomalVRNA
3. DoubleVhelixVofVDNA
4. MitochondrialVDNA
V
14.VAVhikerVexperiencesVmuscleVpainVandVacidosisVwhileVascendingVaVmountainVduringVaVlo
ng,VsteepVclimb.VWhichVisVtheVreasonVforVtheseVmanifestations?
1. CellularVhypoxia
2. Autolysis
3. Heterolysis
4. CellularVedema
V 15.VWhichVfactorVprovidesVDNAVtheVuniqueVmolecularVabilityVtoVreplicate?
1. TheVpairingVofVnitrogenousVbases
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