CRNA interview Questions and Answers
100% Solved
where are alpha 1 receptors located ️️vascular smooth muscle
where are alpha 2 receptors located ️️presynaptic nerve terminals
effects of alpha 1 agonism ️️vasoconstriction, increased svr, increased bp
effects of alpha 2 agonism ️...
CRNA interview Questions and Answers 100% Solved where are alpha 1 receptors located ✔️✔️vascular smooth muscle where are alpha 2 receptors located ✔️✔️presynaptic nerve terminals effects of alpha 1 agonism ✔️✔️vasoconstriction, increased svr, increased bp effects of alpha 2 agonism ✔️✔️inhibition of norepinephrine release How does dexmedetomidine work? ✔️✔️alpha 2 agonism ( blocking release of norepinephrine in brain and spinal cord) Effects of dexmedetomidine ✔️✔️analgesia, decreased catecholamine( hypotension, bradycardia) propofol mechanism of action ✔️✔️potentiation of chloride current mediated channels through the gaba receptor complex propofol effects on CNS ✔️✔️- hypnotic - no analgesia - dec cerebral blood flow and cerebral metabolic rate - dec icp and iop - antiemetic propofol cardio effects ✔️✔️hypotension( vasodilation, dec preload and after load), inhibits Normal baroreflexes( hr response is minimal) respiratory effects propofol ✔️✔️depressant, dec minute ventilation, reduction in upper airway reflexes, mild bronchodilator what could be suspected if pt is tacycardiac on propofol ✔️✔️propofol infusion syndrome( metabolic acidosis) induction dose of propofol ✔️✔️1-2.5mg/kg ( given IV) what do all nmb blocking agents look similar to in terms of chemical structure? ✔️✔️acetylcholine by what mechanism is atracurium eliminated by? ✔️✔️Hoffmans elimination how is neuromuscular blockade monitored ✔️✔️peripheral nerve stimulation( train of four) succinylcholine mechanism of action ✔️✔️binds to cholinergic( nicotinic receptors) to cause depolarization of motor end plate and inhibit neuromuscular transmission (as long as there is adequate amounts of succinylcholine at the site) tof ratio ✔️✔️magnitude of response of fourth twitch compared to the first which kind of neuromuscular blockade will you see fade? ✔️✔️nondepolarizing what kind of neuromuscular blockage will you see all four twitches in a dose related fashion ✔️✔️depolarizing which muscle is last to get paralyzed and first to regain function with nmb? ✔️✔️diaphragm dosing for succinylcholine ✔️✔️0.75-1.5 mg/kg iv what is unique about people with burns and neuromuscular disease as far as acetylcholine receptors? ✔️✔️they have proliferation of exntrajunctional acetylcholine receptors ( can have even more potassium released from cells ) why is potassium increased with succinylcholine? ✔️✔️released from muscles likely from fasciculation's what is risk of using succinylcholine with inhaled anesthetics? ✔️✔️malignant hyperthermia negative effects of succinylcholine ✔️✔️inc iop, increased gastric pressure, hyperkalemia onset of rocuronium ✔️✔️60-120 seconds indications of succinylcholine ✔️✔️rapid intubation( short onset and duration so good for unstable people) contraindications to succinylcholine ✔️✔️myopathies, acute angle glacoma, burns or trauma, family history of MH Flumazenil mechanism of action ✔️✔️binds to gaba a receptors for competitive agonism of Benzos status epilepticus dose of versed ✔️✔️10mg once of 0.2mg/kg once mechanism of action: versed ✔️✔️works on neuronal membrane to increase permeability to chloride ions which increase inhibitory effects of gaba through hyperpolarization
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