The interactionist approach (AO1)
The interactionist approach is a way to explain the development of behaviour in
terms of a range of factors, including both biological and psychological ones.
Factors don’t simply add together but combine in a way that can’t be
predicted by each one separately.
The diathesis-stress model (AO1)
The diathesis-stress model is an interactionist approach to explaining behaviour.
For example, schizophrenia is explained as the result of both an underlying
vulnerability and a trigger, both of which are necessary for the onset of
schizophrenia. Both genes and trauma are seen as diathesis, and stress can
be psychological or biological in nature.
Meehl's model (AO1)
Meehl’s model in the original diathesis-stress model, diathesis was entirely
genetic, the result of a single schizogene. This led to the idea of a biologically
based schizotypic personality, one characteristic of which is the sensitivity to
stress. According to Meehl, if a person does not have the schizogene then
no amount of stress would lead to schizophrenia. However, in carriers of
the gene, chronic stress through childhood and adolescence, in particular
the presence of a schizophrenogenic mother, could result in the development
of the condition.
Modern understanding of diathesis (AO1)
Many genes each appear to increase genetic vulnerability only slightly, there is
no single 'schizogene'. Modern views of diathesis also include a range of factors
beyond the genetic, including psychological trauma; so, trauma becomes the
diathesis rather than the stressor.
Read et al. proposed a neurodevelopmental model in which early trauma
alters the developing brain. Early and severe trauma, such as child abuse,
can seriously affect many aspects of brain development. For example, the
hypothalamic-pituitary-adrenal (HPA) system can become overactive,
making a person much more vulnerable to later stress.
Modern understanding of stress (AO1)
A modern definition of stress includes anything that risks triggering
schizophrenia. Much of the recent research into factors triggering an episode of
schizophrenia has concerned cannabis use. In terms of the diathesis-stress
model, cannabis is a stressor because it increases the risk of
schizophrenia by up to seven times according to dose. This may be because
cannabis interferes with the dopamine system. However, most people do
not develop schizophrenia after smoking cannabis presumably because they
lack the requisite vulnerability factors.
Treatment according to the interactionist model (AO1)
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