NURS 316 Pharmacology Exam 2 Questions and Correct Answers
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Module
NUR 316
Institution
NUR 316
A patient states they have a history of bruising and DVTs. They were taking a drug for this but have now switched to a drug that is safer and doesn't require constant f/u's.
What is the drug and what is the mechanism?
When did they switch to this drug? Apixaban / selective, reversible inhibitor o...
nurs 316 pharmacology exam 2 questions and correct
a patient states they have a history of bruising a
what is the drug and what is the mechanism
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NURS 316 Pharmacology Exam 2
Questions and Correct Answers
A patient states they have a history of bruising and DVTs. They were taking a drug for
this but have now switched to a drug that is safer and doesn't require constant f/u's.
What is the drug and what is the mechanism?
When did they switch to this drug? ✅Apixaban / selective, reversible inhibitor of factor
Xa --> inhibits thrombin-induced platelet aggregation and thrombin generation
After INR < 2.0 after taking Coumadin.
A patient states they have a history of bruising and DVTs. They were taking a drug for
this but have now switched to a drug that is safer and doesn't require constant f/u's.
What education is important?
What should be monitored considering its pharmacokinetics? ✅Take dose if missed,
do not double.
Kidney panels due to excretion in kidney.
A patient is taking a drug for their peptic ulcers with a history of acetaminophen use.
What is the usual oral regimen?
What are side effect(s)?
What history is important and why? What education is important? ✅4/day.
Abd pain and diarrhea.
Gestational due to teratogenic effect. Clients must use contraception.
The drug is misoprostol.
Identify the classes of drugs that suppress RAAS. ✅ACEi's
ARBs
aldosterone antagonists
direct renin inhibitors
,A patient is taking a hypertensive drug that is inhibiting kinase and another enzyme in
the lung.
What is the other enzyme? What does the drug end with?
What electrolyte abnormality(s) could the patient have? ✅ACE, -pril
Low potassium and sodium.
A client is taking a hypertensive drug that is inhibiting kinase and another enzyme.
What is the main side effect from inhibiting kinase? What other side effect(s) could he
have?
What nursing interventions could you educate the client? (5) ✅*Angioedema*, rash,
metallic taste, dry cough.
1) Get up slowly.
2) Fall risk.
3) Report dizziness.
4) Avoid strenuous activity.
5) Client retakes at-home BP.
A client is taking a hypertensive drug that is making him cough. What drug is he on and
what is the next intervention and why? ✅He is on an ACEi.
Switch him to an ARB (Losartan) and tell him to drink water.
ARBs do not cause bradykinin build up.
A client is taking a hypertensive drug that is no longer making him cough.
What drug is this?
What side effects should you educate him on? What education is important? ✅ARB/-
artan
Angioedema, dizziness, hypotension, headache, and insomnia.
1) Take drug in AM.
A client is taking a hypertensive drug that is no longer making him cough.
What electrolyte(s) are going to be lost? ✅Sodium
A client is taking a hypertensive drug. His potassium is now checked to be 5.5.
, What is the possible drug?
What is causing this potassium level?
What side effects should you educate him on? ✅Aldosterone antagonist (Epirenone or
Aldactone, *-one*).
Na and water lost so K stays.
altered mental status, palpitations, muscle twitching, weakness, or paresthesia
A client is having palpitations, muscle twitching, weakness after taking his hypertensive
drug.
What is causing these symptoms?
What should you do (2)? ✅Hyperkalemia due to aldosterone antagonism.
Monitor BUN, creatinine, K.
Do not give salt substitutes (no DASH diet)
A patient is taking a drug after having an MI. This drug relieves pain from low oxygen
levels in the coronary arteries.
What side effects would you educate this patient?
What should you educate the patient then? (3)
What is the drug? ✅Headache, hypotension, tachycardia, dizziness.
1) Check BP before and after. 2) Tell patient to get up slowly.
3) Wear gloves while handling. If not, it can absorb through skin causing vasodilation
and hypotension.
NTG
A patient is taking a drug after having an MI. The drug can be absorbed through the
nurse's skin. What is the mechanism of this drug? What electrolytes are absorbed and
lost? ✅Aldosterone antagonist.
Na and water loss. K is absorbed.
A client is taking a hypertensive drug that directly inhibits renin.
What is the name of the drug?
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