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PRITE – Neuro Exam Questions And Answers

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PRITE – Neuro Exam Questions And Answers Progressive weakness over several days - absent reflexes worse in lower extremities - slow conduction velocity, conduction block Acute inflammatory polyneuropathy Weakness in limbs 2 weeks after a viral gastroenteritis. Weakness in UE/LE, absent DTRs...

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  • October 13, 2024
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PRITE – Neuro Exam Questions And
Answers

Progressive weakness over several days - absent reflexes worse in lower extremities - slow

conduction velocity, conduction block Acute inflammatory polyneuropathy




Weakness in limbs 2 weeks after a viral gastroenteritis. Weakness in UE/LE, absent DTRs.

Spinal fluid shows no cells and elevated protein. Acute inflammatory polyneuropathy




Young pt recovering from flew-like illness w/ progressive weakness and numbness of legs and

feet. Weakness and numbness below middle of thorax. Increased LE DTR's, extensor plantar

reflexes. Urinary incontinence. LP 23 mononuclear cells, protein level 37, nml glucose

Acute transverse myelitis




The single most consistently documented and significant risk factor in the epidemiology of

tardive dyskinesia is? Advanced age




Known risk factors for dementia: Age, family hx, female, Down Syndrome




What psychoactive drug produces amnesia? Alcohol

, PRITE – Neuro Exam Questions And
Answers

What baseline labs should be taken before starting tacrine? ALT and AST (baseline and

f/u)




Clock drawing test is quickly administered and sensitive screen for which d/o?

Alzheimer's




Individuals over 40yo with Down's syndrome frequently develops: Alzheimer's




Amyloid precursor protein in Alzheimer's Disease




Most common cause of dementia: Alzheimer's disease




45 year old with gradual progressive weakness over the past 3-4 months, particularly in the LUE.

Amyotrophic lateral sclerosis

, PRITE – Neuro Exam Questions And
Answers
Atrophy of the intrinsic muscles of the right arm and forearm. Reflexes are generally brisk,

plantar reflexes are extensor. Electrophysiology shows widespread fasciculations, fibrillation and

sharp waves. Dx? Amyotrophic lateral sclerosis




Gradually progressive weakness of legs and dysarthria over months - fasciculations of tongue -

prominent left upper extremity weakness - muscle spasticity - brisk reflexes - normal sensation

Amyotrophic lateral sclerosis




Fasciculations, fibrillations, positive sharp waves on EMG + progressive weakness over several

weeks Amyotrophic lateral sclerosis




70 y/o F sudden onset paralysis R foot and leg. R arm and hand lightly affected. No aphasia or

visual field deficit. Over weeks found with loss bladder control, abulia and lack of spontaneity.

Which vascular area: Anterior cerebral artery (left)




55 y/o hx of weakness and clumsiness x several months. Difficulty w/ fine motor tasks. Arm

muscles twitch and cramp easily, weakening, atrophy. Sensory, coordination, cranial nerve exams

wnl. Underlying illness affects neuronal bodies where? Anterior horn of spinal cord,

medial brainstem and cortex

, PRITE – Neuro Exam Questions And
Answers

Amnesia characterized by loss of memory of events that occur after onset of etiologic condition

or agent Anterograde




Severe spasms and rigidity of limbs intermittently and later more persistent/continuous:

Antiglutamic and anidecarboxylase (anti-GAD) antibodies




Which meds have best results for treating agitation in dementia? Anti-psychotics




Inability to carry out motor activites on verbal command despite intact comprehension & motor

function indicates? apraxia




In managing acute ischemic stroke, administer this within 48 hrs of onset of stroke for beneficial

effect in reducing risk of recurrent stroke, disability and death: Aspirin




An 80yo pt with Alzheimer's is brought in for increasingly combative behavior. Daughter would

like to keep the pt at home if possible. What interventions would be most helpful in this

situation? Assessing for caregiver burnout

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