Summary Robbins Basic Pathology International Edition - Pathology I
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Module
Pathology I
Institution
University Of Nicosia
Book
Robbins Basic Pathology International Edition
This summary document includes keywords to look out for in specific pathological conditions. This covers the cardiovascular system, respiratory system, gastrointestinal system, liver, gallbladder and pancreas! It also entails the different stains and Markers used to differentiate between different ...
Cardiovascular, respiratory, gi, liver, gallbladder and pancreas
November 17, 2024
27
2023/2024
Summary
Subjects
cardiovascular system
cvs
respiratory system
resp
respiratory
cardiovascular
pathology
stains
gastrointestinal
pancreas
liver
gallbladder
hernias
markers
robbins pathology
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CVS:
Keyword (s) Condition Associated
Squatting position, +ve Schamroth sign Tetralogy of Fallot
(clubbing) (Remember: Squatting; in cases of tetralogy of
Fallot -> reduces the resistance and thus
increases pulmonary blood flow -> thus
reducing signs of cyanosis)
“Aschoff” bodies Acute stage of Rheumatic fever
(microscopic feature)
“Fish” mouth Fibrosing stage of Rheumatic fever
(macroscopic feature)
Also seen rheumatic fever endocarditis -
e.g. in the MV
Fusion of commissures + presence of Rheumatic aortic stenosis
triangular orifice (AV)
Myxoid stroma “plastered” onto the aortic AV insufficiency, also characteristic sign of
side of the cusps the usage of fen-phen drug
Tips of papillary muscle insertion is “pearly Tricuspid valve (TV) stenosis
white” (TV)
Pt use drug fen-phen MV insufficiency, AV insufficiency
(NOTE: Glistening appearance as well is
noted on valves of pts taking fen-phen drug;
anti-migraine drug)
“Pitting” oedema Right-sided HF
(Remember: RHF -> thus not enough blood
going to the lungs to pick up O2 -> thus renal
hypoxia -> thus the fluid remains in the
periphery causing fluid retention and oedema ->
“pitting” oedema, ascites etc.)
Endocarditis that affects the right-sided Endocarditis of the carcinoid syndrome
valves; left-sided are rarely involved (Remember: since blood from the GI carcinoids
will enter via the right side of the heart, thus the
right-sided valves will be bathed with the
released mediators; VAPs and amines, of the
carcinoid tumours)
“Spider cells” Rhabdomyoma (Microscopic features)
(Remember: it’s common in children)
“Lepidic” cells Myxoma
“Crescendo” Unstable angina
ST segment elevation, pathological Q wave STEMI
(sometimes T wave inversion is present, but it is
not specific to STEMI)
“Malignant nephrosclerosis” Hyperplastic arterioloscerlosis (because of
malignant hypertension)
“Type A” personality Atherosclerosis (risk factor of developing
atherosclerosis)
“Vulnerable” plaque Unstable atheroslerotic plaque
(Remember: inflammatory cells > smooth
muscle cells, and since there is a greater
amount of inflammatory cells thus there is a
thinner fibrous ap -> acute rupture -> leads to
thrombosis and/or embolism. Also since the lipid
component is greater compared to a stable
plaque -> thus making this plaque unstable)
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