Anoikis Study guides, Revision notes & Summaries

Oncogene - ANSWER-A gene that causes or contributes to the development of cancer Hallmarks of Cancer - ANSWER-Sustaining proliferative signaling Evading growth suppressors Activating invasion and metastasis Enabling replicative immortality Inducing angiogenesis Resisting cell death In vivo oncogenic evidence - ANSWER-Cause cancer when activated in a transgenic animal Render nontumorigenic cells tumorigenic Knock down or knock out in tumorigenic cells renders the cells nontumorigenic...

Oncogene - ANSWER-A gene that causes or contributes to the development of cancer Hallmarks of Cancer - ANSWER-Sustaining proliferative signaling Evading growth suppressors Activating invasion and metastasis Enabling replicative immortality Inducing angiogenesis Resisting cell death In vivo oncogenic evidence - ANSWER-Cause cancer when activated in a transgenic animal Render nontumorigenic cells tumorigenic Knock down or knock out in tumorigenic cells renders the cells nontumorigenic...

Oncogene correct answersA gene that causes or contributes to the development of cancer Hallmarks of Cancer correct answersSustaining proliferative signaling Evading growth suppressors Activating invasion and metastasis Enabling replicative immortality Inducing angiogenesis Resisting cell death In vivo oncogenic evidence correct answersCause cancer when activated in a transgenic animal Render nontumorigenic cells tumorigenic Knock down or knock out in tumorigenic cells renders the ce...

SI Practice Exam 4 |57 Questions with Verified Answers Which of the following statements is FALSE of the spliceosome and splicing mechanism? a. U1 snRNA base pairs with the 5' splice site b. The active spliceosome is produced when U1 and U3 snRNP exit c. U2 snRNA and U6 snRNA base pair to each other for the 1st reaction to occur d. U2 snRNA anneals to the branch site in the intron - CORRECT ANSWER b. The active spliceosome is produced when U1 and U3 snRNP exit Where are SR protein...

codon - ANSWER-set of 3 bases on mRNA that codes for specific amino acid gene expression - ANSWER-degree to which a gene or group of genes is active chromosomes - ANSWER-small structures containing genetic info How many chromosomes/pairs? - ANSWER-46; 23 the 22 pairs that are the same in males/females are called... - ANSWER-autologous Meoisis - ANSWER-when the chromosomes duplicate and cross over -produces 4 gametes with only 1 copy of each chromosome *cross over adds variety a...

Patho Exam 1 Norris, Tommie L. Lippincott CoursePoint for Norris: Porth's Pathophysiology, 10th Edition. Test Bank Atrophy - Ans: When confronted with a decrease in work demands or adverse environmental conditions, most cells are able to revert to a smaller size and a lower and more efficient level of functioning that is compatible with survival. What happens when cells atrophy? - Ans: Reduce their oxygen consumption and other cellular functions by decreasing the number and size of their ...

SI Practice Exam 4 |57 Questions with Verified Answers Which of the following statements is FALSE of the spliceosome and splicing mechanism? a. U1 snRNA base pairs with the 5' splice site b. The active spliceosome is produced when U1 and U3 snRNP exit c. U2 snRNA and U6 snRNA base pair to each other for the 1st reaction to occur d. U2 snRNA anneals to the branch site in the intron - CORRECT ANSWER b. The active spliceosome is produced when U1 and U3 snRNP exit Where are SR protein...

CELL 360 Apoptosis (Exam 3) Terms in this set (11) reasons for going through apoptosis (6) 1. to get rid of unwanted/unneeded cells during development 2. maintaining proper organ size 3. a cell is infected with a virus 4. a cell is no longer needed (ex. during metamorphosis, clearing immune cells after an infection) 5. too much DNA damage 6. the cell is damaged/stressed out apoptosis vs necrosis APOPTOSIS - controlled, systematic - contents of the cell are packa...

A summary of apoptosis, including its intrinsic and extrinsic pathways. Also describes how apoptosis is significant to the development of cancer and what phases are disrupted to create a tumour microenviroment.

Summary of all the lectures of the course Molecular Regulation of Health and Disease (HAP-31806)