Test Bank For Pathophysiology: A Practical Approach 4th Edition By Lachel Story 9781284205435 Chapter 1- 14 Complete Guide .
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Pathophysiology: A Practical Approach
Test Bank For Pathophysiology: A Practical Approach 4th Edition By Lachel Story 5435, 6 , 9967, 3 1 Cellular Function 2 Immunity 3 Hematopoietic Function 4 Cardiovascular Function 5 Respiratory Function 6 Fluid, Electrolyte, and Acid–Base Homeostasis 7 Urinary Function 8 Reproductive Function 9 G...
Test Bank For Pathophysiology A Practical Approach 4th Edition By Lachel Story / BEST STUDY GUIDE
Test Bank for Pathophysiology A Practical Approach 4th Edition by Lachel Story |All Chapters, Year-2023/2024|
Test Bank for Pathophysiology A Practical Approach 4th Edition by Lachel Story |All Chapters, Year-2023/2024|
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Test Bank For Applied Pathophysiology A
h h h h h
Conceptual Approach 4th Edition with
h h h h h
correct QAS 100% verified by experts 2023-
h h h h h h h
2024
h
,Pathophysiology: hA hPractical hApproach: hA hPractical hApproach h4th
hEdition hStory hTest hBank
Chapter h 1 h Cellular h Function
h 1. An h increase h in hcardiac h size h and h function h due h to h increased h workload h is
h termed
A) Atrophy.
B) Functional.
C) Hypertrophy.
D) Inflammation.
h 2. While h assessing h a h patient hdiagnosed h with h Marfan's h Syndrome, h the h nurse
h should
include h which h of hthe h following hthat h is hconsistent h with hthe h syndrome?
A) Cardiac h assessment h for h coarctation h of h the h aorta.
B) Genital h assessment h for h small h testicles.
C) Mental h assessment h for h impairment.
D) Oral h assessment h for h cleft h palate.
h 3. Children h with h PKU h must h avoid h phenylalanine h in hthe h diet. h Phenylalanine h is
h most
likely hto h be h a h component hof
A) Fat.
B) Sugar.
C) Protein.
D) Carbohydrate.
h 4. A h17-year-old h college-bound h student h receives h a h vaccine h against h an h organism
h that
causes hmeningitis. hThis his h an h example h of
A) primary h prevention.
B) secondary h prevention.
C) tertiary h prevention.
D) disease h treatment.
h 5. Metaplasia his
A) The h disorganization h of hcells h into h various h sizes, h shapes, h and h arrangements.
B) The h replacement h of h one h differentiated h cell htype h with h another.
C) The htransformation h of ha h cell h type h to h malignancy.
D) An h irreversible h cellular h adaptation.
, h 6. During ha h routine h ultrasound h of h a h 38 h year h old h women h 26 h weeks h gestation
h reveals h a
fetus hwith ha hsmall h square hhead, hupward h slant h of hthe heyes, hand h low hset
h ears. hThe hnurse h recognizes h that h these h findings h are h consistent h with h which
h of h the h following?
A) Fragile h X hSyndrome.
B) Monosomy hX h (Turner's h Syndrome).
C) Trisomy h21 h (Down's h Syndrome).
D) Trisomy h X h (Klinefelter's h Syndrome).
h 7. While h discussing h treatment h options h with h a h parent h of h a h newly h diagnosed
h Monosomy
X h (Turner's h Syndrome) h child, h the h nurse h should hinclude h which h of hthe
h following?
A) There h is hno htreatment h or hcure.
B) Symptoms h of hthe h condition h are h treated h with h estrogen.
C) Symptoms h of h the h condition h are h treated h with h testosterone.
D) Institutionalization h is h the h preferred h method h of h managing h care.
h 8. Which h of h the h following h assessment h findings h indicates h an h alteration h in
h homeostatic
control h mechanisms?
A) Fever
B) Throat h pain
C) Joint hstiffness
D) Positive h throat h culture
h 9. What h information h should h parents h be h given h about h the h consequences h of
phenylketonuria h (PKU)?
A) Mental h retardation h is h inevitable.
B) PKU h is h commonly h associated h with h other h congenital h anomalies.
C) High h dietary h tyramine h may h help h induce h enzyme h production.
D) Failure h to h treat h properly h results h in h progressive h mental h retardation.
h 10. Injury hthat h occurs h when h blood h flow his h diminished h to htissue h is hcalled injury.
A) hypoxic
B) ischemic
C) hyperemic
D) neoplastic
h 11. Tay hsachs h is h caused h by hwhich h of hthe h following?
A) A hdeficiency h or h absence h of h hexosaminidase hA
B) A hdefect h on h chromosome h 17 h or h 22
C) A hmutation h on h chromosome h 15
, D) An h error h in h converting h phenylalanine h to h tyrosine
h 12. An h obese h but h otherwise h healthy hteen h goes h to ha h health h fair h and h has hher
h blood
pressure h checked. hThis h is han h example h of
A) primary h prevention.
B) secondary h prevention.
C) tertiary h prevention.
D) disease h treatment.
h 13. Characteristics h of h X-linked h recessive h disorders h include h which h of h the
h following?
A) The h son h of h a hcarrier h mother h has ha h 25% hchance h of hbeing haffected.
B) Affected h fathers h transmit h the h gene hto hall h of htheir h sons.
C) All h daughters h of h affected h fathers h are h carriers.
D) Boys h and h girls h are h equally haffected.
h 14. A hfactor h associated h with h risk h of hDown h syndrome h is
A) Maternal hage.
B) Maternal halcohol h intake.
C) Family h history h of h heritable h diseases.
D) Exposure h to hTORCH h syndrome h organisms.
h 15. Which h type h of h gangrene h is h usually h a h result h of harterial h occlusion?
A) Necrosis.
B) Dry.
C) Wet.
D) Gas.
h 16. The h cancer h growth h continuum h is h divided hinto h the h following h stages.
A) Stage h1, h Stage h 2, hStage h3
B) Initiation, h Progression, h Promotion
C) Preliminary, hEvolutionary, hMetastasis
D) Initiation, h Promotion, h Progression
h 17. A hdisease h in h which h the h principal h manifestation h is han h abnormal h growth h of
h cells
leading hto h formation h of htumors his hcalled h a disease.
A) congenital
B) degenerative
C) metabolic
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