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Nur 463 SKM dysrhthmias and critical situations Notes R198,76   Add to cart

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Nur 463 SKM dysrhthmias and critical situations Notes

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This is a comprehensive and detailed note on SKM dysrhthmias and critical situations for Nur 463. *Essential Study Material!!

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  • September 16, 2024
  • 15
  • 2021/2022
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  • Prof. katherine
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Adv. Medsurg Notes– PowerPoint and SK&M
Chapters 7 & 10 – Cardiac Dysrhythmias and Rapid Response teams/Code Blue

OVERVIEW OF CARDIAC PHYSIOLOGY AND ELECTROPHYSIOLOGY
 Physiology
o Specialized cells called pacemaker cells have property of automaticity
 Automaticity  cells can generate a stimulus or an action potential without outside stimulation
 Signal conducted through fibers  generates contraction
o Coordinated electrical activity = cardiac cycle
 1. Impulse generated from pacemaker cells in RA called SA node
 2. Passes through the interndoal tracts and reaches the AV node
 Located in the AV junction  area between atria and ventricles
o AV node has backup system where they can fire an impulse if they do not
receive an impulse from the SA node
 During this time, impulse is slowed for ventricular filling during ventricular diastole (VD)
o Passive ventricular filling – pressure in atria overcomes the pressure of the
valves and the ventricles begin to fill
o Active ventricular filling – atria contract to squeeze blood into ventricles
 3. Impulse is conducted through the bundle of His to ventricles through bundle branches
 Bundle branches = L + R Bundle branch = made up of purkinji fibers
 4. Electrical signal causes atrial systole (contraction)  blood ejects into ventricles
 Atrial kick – extra push of blood into the ventricles to increase CO
 5. Muscle fibers becomes stimulated and cause ventricular systole
 During this time atria are relaxed and begin filling with blood
o Break down of mechanical contraction
 1. Atrial diastole  atrial filling
 All valves closed
 2. Early atrial systole  increase in atrial pressure  opens AV valves
 Ventricles fill
 3. Atrial systole + atrial kick  atrial contraction and emptying
 Ventricles become filled
 4. Early ventricular systole  ventricles begin to contract
 Pressure closes AV valves
 Atria are relaxed
 5. Ventricular systoles  ventricular contraction
 Increased pressure in ventricles
 Aortic and pulmonic valves are open
 Blood ejected into aorta (LV) and pulmonary artery (RV)
 6. Early atrial diastole  ventricles are empty and relax
 Aortic and pulmonary valves are closed
 Cycle repeats from step 1.
THE 12 LEAD EKG
 Leads  looking at the heart from 12 different angles
o 3 standard limb leads (I, II, III)  bipolar leads (positive and negative lead)
 Placed on the arms and legs
 Lead I
 Records magnitude and direction of current flow b/n negative lead on the right arm to
the positive lead on the left arm
o Records flow from right arm to left arm
 Lead II
 Records activity b/n negative lead on right arm and the positive lead on the left leg

, o Records flow from right arm to left leg
 Normal ECG wave forms are upright in these leads, lead II produces most upright
waveform
 Goes through the largest part of the heart  better picture d/t more electrical activity
 Lead III
 Records activity from the negative lead on the left arm to the positive lead on the left leg
o Records flow from left arm to left leg
 Not the same degree of amplitude as you would in lead II, but still get a good picture
o You would prefer this lead dependent on the damage to the heart (EX:
cardiomyopathy)
 The LV might shift left so a better picture would be to the left side
o Also dependent on the shape of the heart and where it is in the chest wall
o 3 augmented limb leads (aVR, aVL, aVF)  unipolar (record electrical flow in only one direction)
 From heart out to the body
 aVR  from heart to right arm
 aVL  form heart to left foot
 aVF  from heart to left arm
o A stands for augmented d/t small ECG complexes (they must be augmented or
enlarged)
o V stands for voltage
o Subscripts R, L, and F stand for location (right arm, left arm, left foot)
 Where the electrode is located
 They are like a mirror image of the other types of leads
 Provide confirmation of standard leads
o 6 precordial leads (V1, V2, V3, V4, V5, V6)  chest leads that are unipolar
 Placed over the heart (do not need to know locations, just for own reference)
 V1  4th intercostal space (ICS), right sternal boarder (RSB)
o Major lead for dysrhythmia monitoring
 Will be used in tandem with lead III
 V2  4th ISC LSB
 V3  halfway between V2 and V4
o Best to recognize ischemic changes in the heart
 Used in tandem with lead III for myocardial ischemia for at risk pt
 Acute coronary syndrome, risk for silent ischemia, recent cardiac
interventions
th
 V4  5 ICS, LMCL (midclavicular line)
th
 V5  5 ICS, LAAL (anterior axillary line)
 V6  5th ICS, LMAL (midaxillary line)
 Offer a good picture of ventricular activity
 Unipolar  positive electrode and the AV node as central reference
 Impulses direction
o Toward electrode = positive QRS complex
o Away from electrode = negative QRS complex
CARDIAC MONITORING
 Telemetry
o Most ICUs use a 5-lead system
 Limb leads are placed (I, II, III)
 One precordial lead is placed  can be moved for different views
o Most ERs use a three lead system
 Limb leads I, II, III
 RA and LA leads are placed just above the clavicles
 LL lead is placed on the left abdominal area below umbilicus

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