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OCCTH 1 PART 2 (WEEK 3 - neuromotor impairments) Questions & Answers Already Passed!!

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Motor Neglect - ️️- impaired initiation or execution of movement into contralateral hemispace by either limb - often difficult to distinguish between sensory or motor neglect ◦Limb akinesia: failure to move limb ◦Hypokinesia: limb moves only after long delay and much encouragement ◦hypometria: movements are of decreased amplitude ◦impersistence: inability to maintain a movement or posture ◦motor perseveration: inability to disengage from a motor activity ◦motor extinction: delay or failure to move contralateral limb when also required to move ipsilateral limb LESIONS IN CORTEX// APRAXIA - ️️inability to perform goal-directed motor activity; omissions, disturbed sequence, clumsiness, perseveration, inability to gesture or use common objects 4 types: IDEOMOTOR: Errors made with goal-directed movements; improves with use of actual movements (Awkward or clumsy use of tools to complete tasks; may not be able to complete movement pattern for brushing teeth, but able to complete task in context with real objects) IDEATIONAL(CONCEPTUAL): Error in use of common object use and sequencing of multistep tasks (may attempt to use toothbrush to brush hair) CONSTRUCTIONAL: Difficulty with copying, drawing, and constructing 2D and 3D designs (setting table, making sandwich, etc.) DRESSING: inability to dress oneself Neuromaturational Theory of motor development - ️️Changes in neural structures is directly related to changes in motor function - Changes in neural structures -- Changes in motor function - Changes are due to interaction of multiple systems & contexts - CNS damage can lead to regression to lower levels and stereotypical behaviors Assumptions of Therapeutic Approaches - ️️- CNS is hierarchically organized- Motor control is heterarchical system (smaller subsystems have effect on overall motor function) - Environment, Activity demands, and client factors all relate to performance - Behavioral changes reflect attempts to compensate and achieve task performance ◦use remaining capacities in addition to new strategies to continue functional performance (ex: learned non-use) site, extent - ️️______ and _________ of CNS damage will determine the type of motor dysfunction ** damage in specific locations will correlate to damage/deficits PATHWAY of MOTOR messages (start in neuro system and move to skeletal) - ️️Precentral Gyrus: primary motor cortex (combines with sensory info here) ---- internal capsule ---- thalamus ---- brainstem ----- spinal cord (ventral horn to ventral root to PNS) ---- skeletal muscle LESIONS IN CORTEX// PRECENTRAL GYRUS - ️️loss of voluntary muscle movement on contralateral side ◦spasticity, hyperreflexia LESION IN CORTEX// HYPOTONIA, HYPERTONIA, SPASTICITY - ️️Hypotonia: decreased muscle tone or flaccidity; less than normal resistance to passive elongation Hypertonia: increased muscle tone; more than normal resistance to passive elongation Spasticity: *velocity-dependent* increase in tonic stretch reflexes and exaggerated tendon reflexes ◦component of hypertonicity ***Change in tone can be caused by both mechanical or neurological factors of the muscle Mechanical factors: changes in the elastic part of the muscle Neurological factors: spasticity in muscle because of damage to the brain LESIONS IN CORTEX// CLONUS - ️️oscillating contraction and relaxation of a limb caused by alternating stretch reflex and inverse stretch reflex of spastic muscle - Caused by hyperreflexia of muscleLESIONS IN CORTEX// LEAD PIPE RIGIDITY - ️️hypertonus in agonist and antagonist muscles, resistance to movement not velocity-dependent, but felt throughout ROM LESIONS IN CORTEX// LOSS OF FRACTIONATION AND WEAKNESS - ️️FRACTIONATION: inability to move a single joint without producing unnecessary movements in other joints; causing stereotyped movement patterns (difficulty w/ small movements) WEAKNESS: inability to generate necessary force for effective motor action Basal Ganglia - ️️Integration and storage of stereotypic and automatic motor patterns ◦Learned motor patterns that require little conscious cortical input ◦Dressing, walking, hand to mouth, etc. Tone and posture Initiation and dis-inhibition of movement Error correction during movement

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Institution
OCCTH 1- Neuromotor Impairments)
Course
OCCTH 1- neuromotor impairments)

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OCCTH 1 PART 2 (WEEK 3 - neuromotor
impairments)
Motor Neglect - ✔️✔️- impaired initiation or execution of movement into contralateral
hemispace by either limb

- often difficult to distinguish between sensory or motor neglect
◦Limb akinesia: failure to move limb
◦Hypokinesia: limb moves only after long delay and much encouragement
◦hypometria: movements are of decreased amplitude
◦impersistence: inability to maintain a movement or posture
◦motor perseveration: inability to disengage from a motor activity
◦motor extinction: delay or failure to move contralateral limb when also required to move
ipsilateral limb

LESIONS IN CORTEX// APRAXIA - ✔️✔️inability to perform goal-directed motor
activity; omissions, disturbed sequence, clumsiness, perseveration, inability to gesture
or use common objects
4 types:
IDEOMOTOR: Errors made with goal-directed movements; improves with use of actual
movements (Awkward or clumsy use of tools to complete tasks; may not be able to
complete movement pattern for brushing teeth, but able to complete task in context with
real objects)

IDEATIONAL(CONCEPTUAL): Error in use of common object use and sequencing of
multistep tasks (may attempt to use toothbrush to brush hair)

CONSTRUCTIONAL: Difficulty with copying, drawing, and constructing 2D and 3D
designs (setting table, making sandwich, etc.)

DRESSING: inability to dress oneself


Neuromaturational Theory of motor development - ✔️✔️Changes in neural structures is
directly related to changes in motor function

- Changes in neural structures --> Changes in motor function

- Changes are due to interaction of multiple systems & contexts

- CNS damage can lead to regression to lower levels and stereotypical behaviors

Assumptions of Therapeutic Approaches - ✔️✔️- CNS is hierarchically organized

, - Motor control is heterarchical system (smaller subsystems have effect on overall motor
function)

- Environment, Activity demands, and client factors all relate to performance

- Behavioral changes reflect attempts to compensate and achieve task performance
◦use remaining capacities in addition to new strategies to continue functional
performance
(ex: learned non-use)

site, extent - ✔️✔️______ and _________ of CNS damage will determine the type of
motor dysfunction

** damage in specific locations will correlate to damage/deficits

PATHWAY of MOTOR messages (start in neuro system and move to skeletal) -
✔️✔️Precentral Gyrus: primary motor cortex (combines with sensory info here) ---->
internal capsule ----> thalamus ----> brainstem -----> spinal cord (ventral horn to ventral
root to PNS) ----> skeletal muscle

LESIONS IN CORTEX// PRECENTRAL GYRUS - ✔️✔️loss of voluntary muscle
movement on contralateral side
◦spasticity, hyperreflexia

LESION IN CORTEX// HYPOTONIA, HYPERTONIA, SPASTICITY - ✔️✔️Hypotonia:
decreased muscle tone or flaccidity; less than normal resistance to passive elongation

Hypertonia: increased muscle tone; more than normal resistance to passive elongation

Spasticity: *velocity-dependent* increase in tonic stretch reflexes and exaggerated
tendon reflexes
◦component of hypertonicity

***Change in tone can be caused by both mechanical or neurological factors of the
muscle
Mechanical factors: changes in the elastic part of the muscle
Neurological factors: spasticity in muscle because of damage to the brain

LESIONS IN CORTEX// CLONUS - ✔️✔️oscillating contraction and relaxation of a limb
caused by alternating stretch reflex and inverse stretch reflex of spastic muscle

- Caused by hyperreflexia of muscle

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Institution
OCCTH 1- neuromotor impairments)
Course
OCCTH 1- neuromotor impairments)

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Uploaded on
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Number of pages
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Written in
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R174,29
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